Prohibitin Monoclonal Antibody, 100ul Cell Separation and Collection Mutations in EPHA2 are the

Mutations in EPHA2 are the cause of certain genetically-related cataract disorders

a stimulator of macroautophagy

and two MAGE domains in the C-terminus

The phosphorylation state of this kinase in synaptic terminals was shown to be regulated by membrane depolarization via calcineurin

spinal cord and adrenal gland

Prohibitin Monoclonal Antibody, 100ul Cell Separation and Collection Mutations in EPHA2 are theThis gene is evolutionarily conserved, and its product is proposed to play a role in human cellular senescence and tumor suppression. Antiproliferative activity is reported to be localized to the 3' UTR, which is proposed to function as a trans acting regulatory RNA. Several pseudogenes of this gene have been identified. Alternative splicing results in multiple transcript variants.