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insulin-resistant individuals

results in multiple transcripts encoding different isoforms

whereas the alpha2C subtype modulated neurotransmission at lower levels of nerve activity

Alternative splicing of downstream exons is also observed| which results in different forms of the stimulatory G-protein alpha subunit| a key element of the classical signal transduction pathway linking receptor-ligand interactions with the activation of adenylyl cyclase and a variety of cellular reponses

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